TORONTO, Sept. 20 PRNewswire – The US Centers for Disease Control (CDC) yesterday presented data at the Interscience Conference on Antimicrobial Agents and Chemotherapy (ICAAC) that supports data presented earlier by Dr. Bart Classen, an immunologist at Classen Immunotherapies, proving vaccines cause insulin dependent diabetes.
Earlier this month, Dr. Bart Classen presented data at the International Public Conference on Vaccination which proved vaccines are the largest cause of insulin dependent diabetes in children. This data included data from a prospective randomized clinical trial in Finland showing vaccinated groups had a statistically significant, 17%, increased risk of diabetes after 10 years follow up. Further analysis of people receiving newer, more potent, hemophilus vaccine indicated that these hemophilus vaccines increased the risk of diabetes by about 25%. The CDC’s study indicated that 247 of 260 diabetics received the hemophilus vaccine compared to 733 of 780 controls. This indicates the hemophilus vaccines associated with an odds ratio of 1.22 or an approximately 22% increased risk of
diabetes, almost identical to what Classen found.
Dr. Classen has published both animal and human data with several different vaccines that immunization starting in the first month of life was associated with an decreased risk of diabetes while immunization starting after the second month of life is associated with an increased risk of diabetes. Classen has published data from both New Zealand and Italy that the Hepatitis B vaccine, when given after 2 months of life, is associated with an approximately 50% increased risk of diabetes. The CDC only published part of their data on the hepatitis B vaccine. The CDC found the hepatitis B vaccine was associated with an overall decreased risk of diabetes (relative risk 0.92) which is consistent with an large per cent of those vaccinated receiving the vaccine at birth. The CDC however found that those immunized starting after 2 months of life were at a 60% increased risk of developing diabetes than those immunized starting in the first month of
life (.88/.52). The CDC’s hepatitis B vaccine data is thus also consistent with Classen’s finding.
The CDC’s study and analysis suffered from some obvious limitations and flaws. The CDC studied only 260 diabetics and 780 controls while Dr. Classen’s studies typically have involved 100,000 people or more. The CDC’s study did not compensate for the interaction between the two different vaccines since people received both the hepatitis B vaccine and the hemophilus vaccine while Classen studied these vaccines separately. The CDC study was also limited because over 94% of controls were vaccinated with the hemophilus vaccine while Classen performed studies where almost none of the controls were vaccinated. The net effect is the CDC’s study did not have the power of Classen’s studies. More importantly the CDC’s analysis was flawed because the results were altered, after they were calculated, to compensate for a family history of diabetes. This practice that is considered unorthodox in part because the CDC has many different “fudge” factors by which it can manipulate the results. Last year the CDC presented data from the same data HMO data source but manipulated their results using a different variable to compensate for breast feeding. In 1997 the CDC also presented an analysis on the hepatitis B vaccine, also from the same HMO data source, but did not use either “fudge” factor. In this study the hepatitis B vaccine, when given after 8 weeks of life, was associated with a
90% increased risk of diabetes. The fact that the CDC manipulates similar data in different years using different “fudge” factors has raised suspicion that their analysis is severely flawed and their interpretations of the data
should be viewed with caution.
Dr. Bart Classen presented data at the International Public Conference on Vaccination on September 10 that vaccines cause approximately 80% of cases of insulin dependent diabetes in children who have received multiple vaccines starting after 2 months of life. Children receive 10 or more vaccines and many of these are associated with an increased risk of diabetes. Classen’s data and other published data indicates the following vaccines are associated with an increased risk of diabetes (increased risk): hepatitis B (50%), hemophilus (25%), tetanus (20%), diphtheria (9%), pertussis (25%), mumps- rubella (23%). These findings are supported by a case control study performed in Europe. The cumulative effect of all these vaccines on diabetes is tremendous. Highly immunized sailors in the US navy have been found to develop insulin dependent diabetes at a rate of 5.5 times that of controls even though their rate of diabetes on entering the navy was equal to that of controls.
The US and other governments provide compensation for vaccine induced injuries however there is a statute of limitations. Insulin dependent diabetes cost the patient about $1 million over their lifetime. Many diabetics have contacted Dr. Classen about receiving compensation from the US government. Information on this subject can be found on the Vaccine Safety Website (http://vaccines.net).
Childhood Vaccinations and Juvenile-Onset (Type-1) Diabetes
by Harris Coulter, Ph.D.
President, Center for Empirical Medicine Testimony before the Congress of the United States, House of Representatives, Committee on Appropriations, subcommittee on Labor, Health and Human Services, Education, and Related Agencies April 16, 1997
Diabetes, both juvenile-onset (Type I) and adult-onset (Type II), is a major health problem in the United States, and the number of diabetics is increasing every year. In 1947, there were an estimated 600,000 cases of diabetes in the United States.(1)
Thirty years later, in 1976, Henry Bearn wrote: It is perhaps not generally appreciated that in the United States diabetes, or at least the recognition of the disease, has increased about 300 percent over the last fifteen years. It is the second leading cause of blindness, and the third cause of death. In 1950 there were 1.2 million diabetics in the United States; the estimation now is that there are over 10 million, yet the population has increased by only 50 percent.(2)
Today the Metropolitan Life Insurance Co.’s quarterly Statistical Bulletin estimates that diabetics make up 5 percent of the US population, or 13 million persons.(3) Of these, 85-90 percent are adult onset, which is more or less controlled by diet and exercise; the other 10-15 percent require daily injections of insulin. So, while the US population has approximately doubled since the 1940’s, the number of diabetics has risen more than 20 times. While the statistical data, like any medical statistics, are based to some degree on estimates, there has clearly been a huge increase in the number of diabetics in the United States. Billions Spent to Help Diabetics – Furthermore, diabetics consumer about 15 percent of all health care costs, again according to Metropolitan Life.
People not only die from diabetes (160,000 cases in 1994) but the disease leads to cardiovascular complications, stroke, gangrene of the extremities requiring amputation, kidney failure, and blindness. With an estimated total health bill in the United States of about $1 trillion per year at the end of the 20th century, the annual bill for the care and treatment of diabetics will shortly amount to $100-$150 billion unless steps are taken to prevent this. If the Medicare and Medicaid expenditures for treatment of diabetics could be reduced by half, it would be a major savings.
African Americans At Risk – Of particular concern is the heightened prevalence of diabetes in the American black population. In 1991 the death rate from diabetes in American white males was 11.5/100,000 (resident population), for white females it was 9.6; for black males it was 24.6 and for black females it was 25.7. In other words, the death rate for blacks is 2-3 times as high as for whites (4).
This is an especially serious problem in the armed services. The expected incidence of Type-I (insulin-dependent) diabetes for persons aged 17-34 is 4/100,000 for whites and 90/100,000 for black sailors in the 17-34 age group. (5) The authors of this study admit ignorance about the reason why the diabetes incidence should be higher in black naval personnel. Especially worrisome in this connection, is the ignorance of scientists about the reasons for the steep rise in diabetes. It may be due, in part, to earlier diagnosis or better treatment of the disease, thus preventing or postponing death and/or the development of stroke, kidney failure, and blindness. But this factor cannot account for the tremendous increase in cases since the 1940s. Genetic and Environmental Factors – In any case, the very origin of diabetes is still a mystery. Since the late 19th century, diabetes has been known to be related to the pancreas and, in 1922, Canadians Frederick Banting and Charles H. Best, discovered that the missing factor was insulin – an internal secretion of the pancreas. But why does the pancreas stop, or fail to start, secreting insulin? Or, more specifically, why do the beta-cells of the pancreas cease to perform their functions? The consensus on the causation of diabetes was expressed in 1976 in a paper by Alexander Bearn: “Diabetes appears to be one of those diseases in which susceptibility may be inherited but where environmental factors may lead to the onset of disease and illness.” (6) One environmental factor – viral infection – has been recognized; the other factor of significance for diabetes is the presence of an autoimmune process. (7) But the cause or causes of the epidemic of autoimmune disease in the United States, which commenced in the 1950’s, are themselves mysterious. (8) Since the incidence and prevalence of diabetes continues to rise at a rather rapid rate in the United States and the other industrialized countries, every possible causal or environmental factor is worth examining. On such factor which has hardly been investigated at all is the relationship with childhood vaccinations.